REGULATION OF RASSF BY NON-CODING RNAS IN DIFFERENT CANCERS: RASSFS AS MASTERMINDS OF THEIR OWN DESTINY AS TUMOR SUPPRESSORS AND ONCOGENES

Regulation of RASSF by non-coding RNAs in different cancers: RASSFs as masterminds of their own destiny as tumor suppressors and oncogenes

Ras-association domain family (RASSF) proteins are tumor suppressors and have gained phenomenal limelight because of their mechanistic role in the prevention/inhibition of Puzzle carcinogenesis and metastasis.Decades of research have demystified wide ranging activities of RASSF molecules in multiple stages of cancers.Although major fraction of RASS

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Designing technologies for self-care: Describing the lived experiences of individuals with rheumatoid arthritis

Self-care of a chronic illness is a lifelong management process that includes taking medications, monitoring symptoms, and coping with emotional and lifestyle changes.Human-Computer Interaction (HCI) research has often responded to these needs by developing Video Cards technologies that help an individual quantify aspects of their chronic illness,

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Dysregulated SREBP1c/miR-153 signaling induced by hypertriglyceridemia worsens acute pancreatitis and delays tissue repair

Severe acute pancreatitis (AP) is a life-threatening disease with up to 30% mortality.Therefore, prevention of AP aggravation and promotion of pancreatic regeneration are critical during the course and treatment of AP.Hypertriglyceridemia (HTG) is an established aggravating factor for AP that hinders pancreatic regeneration; however, its exact mech

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